Advertisement

Editorial

  • Fran Balkwill
    Correspondence
    Tel.: +44 20 7882 6108; fax: +44 20 7882 6110.
    Affiliations
    Cancer Research UK, Translational Oncology Laboratory, Barts and The London, Queen Mary’s School of Medicine & Dentistry, John Vane Science Centre, Charterhouse Square, London EC1M 6BQ, United Kingdom
    Search for articles by this author
      The idea that cancer and inflammation are linked is not new, but has become more prominent in the past ten years. During this time, a number of seminal papers have changed our ideas about the role of the inflammatory mediators and infiltrating host cells of the innate immune system in the cancer microenvironment. These papers are supported by definition of the cytokine and chemokine network and identification of leucocyte subsets that contribute to chronic inflammation. A study of inflammation and cancer also provides an intellectual context for understanding a fact that has been largely ignored during dissection of the genetic basis of cancer, i.e., that cancers are not only composed of genetically altered malignant cells.
      To read this article in full you will need to make a payment

      Purchase one-time access:

      Academic & Personal: 24 hour online accessCorporate R&D Professionals: 24 hour online access
      One-time access price info
      • For academic or personal research use, select 'Academic and Personal'
      • For corporate R&D use, select 'Corporate R&D Professionals'

      Subscribe:

      Subscribe to European Journal of Cancer
      Already a print subscriber? Claim online access
      Already an online subscriber? Sign in
      Institutional Access: Sign in to ScienceDirect